Maintenance of GABAergic Activity by Neuregulin 1-ErbB4 in Amygdala for Fear Memory

(Neuron 84, 835–846; November 19, 2014) In the published paper, funding from the National Natural Science Foundation of China was incorrectly attributed to Lin Mei. The corrected acknowledgments section is included here. The authors apologize for this error. This work is supported in part by grants from the NIH (L.M. and W.-C.X.), National Alliance for Research on Schizophrenia and Depression (NARSAD) (L.M., Y.L., and D.-M.Y.), National Natural Science Foundation of China (81329003, U1201225, 31430032, 31271171, and 81471116 to T.-M.G. and B.-M.L.), Guangzhou Science and Technology Project (7411802013939 to T.-M.G.), and Program for Changjiang Scholars and Innovative Research Team in University (IRT1142 to T.-M G.). L.M. is a NARSAD Distinguished Investigator. Y.L. and D.-M.Y. were NARSAD Young Investigators. We thank Dr. Hongjun Song for lentivirus vectors; Dr. Kathleen Yee and Dr. Martin Gassmann for ErbB4 null mutant mice; Dr. Alvin Terry and Dr. Patrick Callahan of the Small Animal Behavioral Core at Georgia Regents University for assistance in behavioral analysis; and Dr. Alexis Stranahan for advice and assistance in stereology analysis. Maintenance of GABAergic Activity by Neuregulin 1-ErbB4 in Amygdala for Fear MemoryLu et al.NeuronOctober 23, 2014In BriefLu et al. show that neuregulin 1 (NRG1) and ErbB4 are critical for maintaining GABAergic activity in amygdala and are necessary for fear memory. As NRG1 and ErbB4 are susceptibility genes of schizophrenia, these results shed light on potential pathophysiological mechanisms. Full-Text PDF Open Archive