Integrated bioinformatics analysis reveals significant genes associated with cryptorchidism (CO)

基因 生物 古伯纳库姆 基因本体论 基因调控网络 细胞外基质 折叠变化 遗传学 生物信息学 计算生物学 基因表达 内分泌学
作者
Liren Hu,Xianming Fan,Jianfeng Lin,Fulv Liang,Jianping Tu,Zhaojian Guo
出处
期刊:Research Square - Research Square
标识
DOI:10.21203/rs.3.rs-1690590/v1
摘要

Abstract Objective: we performed a comprehensive bioinformatics analysis to identify potential genes and signaling pathways associated with cryptorchidism. Materials and Methods: This study aimed to identify genes significantly associated with cryptorchidism (CO), between the LE and ORL gubernaculum groups at GD17 and GD19 stages, using a GSE57924 dataset. We identified other DEGs which were changed to up-regulation or down-regulation between the groups by adjusting the cut-off (|log2 fold change (FC)| ≥0.5 and adjusted p <0.05). We subjected the identified DEGs to gene ontology (GO) and pathway enrichment analyses, using an online database DAVID, then employed Cytoscape v3.7.1 to construct a protein-protein interaction network. Results: We detected functional hub genes using MCODE. Overall, we identified 147 DEGs between the LE gubernaculum group and ORL gubernaculum group, of which 33 genes were up-regulated (21 genes always down-regulated and 13 genes from down-regulated in GD17 to up-regulated in GD19) while 96 was down-regulated (27 genes always down-regulated and 69 genes from up-regulated in GD17 to down-regulated in GD19). Gene Ontology (GO) revealed that cell adhesion and extracellular matrix were the most abundant biological processes and Cellular Components, whereas the ECM-receptor interaction was the most significant pathway. Analysis of hub genes in the PPI network revealed that 7 genes including Igfbp5, Cyr61, Lamb1, Lamc1, Cp, Fn1, and Ltbp1, were significantly associated with CO. Conclusions: we successfully identified 7 significant markers that provide valuable insights into the molecular mechanism of cryptorchidism. Of note, one of the genes (Fn1) was verified significant differential expression gene in human testicular cancer.

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