Strain Magnitude-Dependent Calcific Marker Expression in Valvular and Vascular Cells

钙化 拉伤 免疫染色 主动脉瓣 瓣膜性心脏病 骨形态发生蛋白2 血管平滑肌 内科学 血管组织 信使核糖核酸 生物 病理 细胞生物学 内分泌学 免疫组织化学 医学 体外 平滑肌 基因 生物化学 植物
作者
Zannatul Ferdous,Hanjoong Jo,Robert M. Nerem
出处
期刊:Cells Tissues Organs [Karger Publishers]
卷期号:197 (5): 372-383 被引量:16
标识
DOI:10.1159/000347007
摘要

Aortic valve disease and atherosclerosis tend to coexist in most patients with cardiovascular disease; however, the causes and mechanisms of disease development in heart valves are still not clearly understood. To understand the contributions of the magnitude of cyclic strain (5% hypotension, 10% physiological, and 15% hypertension) in calcification, we used a model system of tissue-engineered collagen gels containing human aortic smooth muscle cells and human aortic valvular interstitial cells, both isolated from noncalcific heart transplant tissue. The compacted collagen gels were cultured in osteogenic media for 3 weeks in a custom-designed bioreactor and all assessments were performed at the end of the culture period. The major finding of this study is that bone morphogenic protein (BMP)-2 and BMP-4 and transforming growth factor-β1 mRNA expression significantly changed in response to the magnitude of applied strain in valvular cells, while the lowest expression was observed for the representative physiological strain. On the other hand, mRNA expression in vascular cells did not vary in response to the magnitude of strain. Regarding BMP-2 and BMP-4 protein expression determined by immunostaining, trends were similar to mRNA expression in vascular and valvular cells, where only valvular cells showed a varied protein expression depending on the magnitude of the strain applied. Our results suggest that cellular differences exist between vascular and valvular cells in their response to altered levels of cyclic strain during calcification.

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