脱色
医学
皮肤病科
光环
白癜风
痣
黑色素细胞痣
病理
黑色素瘤
癌症研究
量子力学
银河系
物理
作者
Emi Yamazaki,Moyuka Irimada,Rui Sasaki,Kenichiro Tsuchiyama,Kenshi Yamasaki,Setsuya Aiba
标识
DOI:10.1111/1346-8138.15643
摘要
Figure S1. Improvement of the depigmentation after the resection of the original halo nevus. Clinical images of the first visit (before resection) and 7 weeks after the resection of the original halo nevus (14 weeks after the first visit) are shown: (a) neck, (b) upper back, (c) the resected original nevus in the right forearm and (d) left lower leg. Blue and red arrows indicate locations of halo nevi and vitiligo, respectively. Figure S2. Histopathological analysis of interleukin (IL)-17 and C-X-C motif chemokine (CXCL)10 expression in the intradermal nevus without Sutton's phenomenon. Three intradermal nevus specimens were immunofluorescence-stained with anti-IL-17 antibody (AF-317-NA; R&D Systems, Minneapolis, MN, USA) or with anti-CXCL10 antibody (LS-c137619; LifeSpan BioSciences, Seattle, WA, USA). The location of IL-17-positive cells or CXCL10-positive cells are shown by green fluorescence (Alexa Flour® 488, Thermo Fisher Scientific K.K., Tokyo, Japan). Nuclei are visualized with 4′,6′-diamidino-2-phenylindole dihydrochloride. Bars, 200 μm. Table S1. Halo nevi associated with Turner syndrome Please note: The publisher is not responsible for the content or functionality of any supporting information supplied by the authors. Any queries (other than missing content) should be directed to the corresponding author for the article.
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