Uncoupling IL-2 Signals that Regulate T Cell Proliferation, Survival, and Fas-Mediated Activation-Induced Cell Death

受体 流式细胞术 突变体 生物 T细胞 细胞生物学 收回 分子生物学 免疫学 免疫系统 遗传学 基因 数学 纯数学
作者
Luk Van Parijs,Yosef Refaeli,James D. Lord,Brad H. Nelson,Abul K. Abbas,David Baltimore
出处
期刊:Immunity [Cell Press]
卷期号:30 (4): 611-611 被引量:22
标识
DOI:10.1016/j.immuni.2009.04.002
摘要

(Immunity 11, 281–288; September 1, 1999) The authors have agreed to retract this paper because of duplication of the flow cytometry dot plots in Figure 1C (labeled as WT and Δ355+8F) by the first author. This figure shows the reconstitution of mouse cells deficient in IL-2 receptor beta with various wild-type and mutant human IL-2 receptor beta chains. This data established the system used to test various properties of these receptor chains in the later work of the paper. This matter was investigated by the California Institute of Technology and the Office of Research Integrity at the United States Department of Human Services, which found the first author to be responsible for the scientific misconduct pertaining to the issue described here. The authors stand by the validity of the other figures and sincerely apologize for the inconvenience caused by this retraction. RETRACTED: Uncoupling IL-2 Signals that Regulate T Cell Proliferation, Survival, and Fas-Mediated Activation-Induced Cell DeathVan Parijs et al.ImmunitySeptember 01, 1999In BriefThis article has been retracted at the request of the authors. Please see Elsevier Policy on Article Withdrawal ( http://www.elsevier.com/locate/withdrawalpolicy ). Reason: The authors have agreed to retract the paper because of duplication of the flow cytometry dot plots in Figure 1C (labeled as WT and Δ355+8F) by the first author. This figure shows the reconstitution of mouse cells deficient in IL-2 receptor beta with various wild type and mutant human IL-2 receptor beta chains. This data established the system used to test various properties of these receptor chains in the later work of the paper. Full-Text PDF Open Archive

科研通智能强力驱动
Strongly Powered by AbleSci AI
科研通是完全免费的文献互助平台,具备全网最快的应助速度,最高的求助完成率。 对每一个文献求助,科研通都将尽心尽力,给求助人一个满意的交代。
实时播报
2秒前
传奇3应助weiweiwei采纳,获得10
3秒前
失眠孤兰关注了科研通微信公众号
3秒前
自由的云朵完成签到 ,获得积分10
3秒前
3秒前
缓慢听安完成签到,获得积分10
4秒前
halo发布了新的文献求助10
6秒前
共享精神应助HolmeTao采纳,获得10
6秒前
6秒前
成就青荷应助科研通管家采纳,获得10
7秒前
思源应助科研通管家采纳,获得10
7秒前
香蕉觅云应助科研通管家采纳,获得10
7秒前
CodeCraft应助科研通管家采纳,获得10
7秒前
orixero应助科研通管家采纳,获得10
8秒前
布蕾脆脆应助科研通管家采纳,获得10
8秒前
充电宝应助科研通管家采纳,获得10
8秒前
温一完成签到 ,获得积分10
8秒前
Liulu发布了新的文献求助10
8秒前
斯文败类应助科研通管家采纳,获得10
8秒前
香蕉觅云应助科研通管家采纳,获得10
8秒前
李健应助科研通管家采纳,获得10
8秒前
9秒前
9秒前
9秒前
9秒前
小马甲应助科研通管家采纳,获得10
9秒前
9秒前
cdercder应助科研通管家采纳,获得10
9秒前
斯文败类应助科研通管家采纳,获得10
10秒前
地球发布了新的文献求助10
10秒前
李满际完成签到,获得积分10
10秒前
無影丶发布了新的文献求助10
10秒前
陈陈完成签到 ,获得积分20
13秒前
豆包发布了新的文献求助10
14秒前
weiweiwei完成签到,获得积分10
16秒前
张欢馨应助halo采纳,获得10
18秒前
小二郎应助369ninja采纳,获得10
18秒前
钱来完成签到,获得积分10
20秒前
20秒前
激动的55完成签到 ,获得积分10
21秒前
高分求助中
(应助此贴封号)【重要!!请各用户(尤其是新用户)详细阅读】【科研通的精品贴汇总】 10000
APA handbook of comparative psychology: Basic concepts, methods, neural substrate, and behavior 1000
Health Psychology 1000
全员动态考核,锚定高质量发展:读懂同济大学教师人事改革新政的深层价值 900
Matrix Methods in Data Mining and Pattern Recognition Second Edition 510
The fast track to determining transfer functions of linear circuits: The student guide 500
Römisch-Germanische Forschungen 500
热门求助领域 (近24小时)
化学 材料科学 医学 生物 纳米技术 工程类 有机化学 化学工程 生物化学 计算机科学 内科学 物理 复合材料 催化作用 细胞生物学 无机化学 光电子学 物理化学 电极 基因
热门帖子
关注 科研通微信公众号,转发送积分 7595620
求助须知:如何正确求助?哪些是违规求助? 9172266
关于积分的说明 19634758
捐赠科研通 7172848
什么是DOI,文献DOI怎么找? 3267840
关于科研通互助平台的介绍 2432659
邀请新用户注册赠送积分活动 2260962