摘要
articleATP synthesis by oxidative phosphorylationA. E. SeniorA. E. SeniorDepartment of Biochemistry, University of Rochester Medical Center,New York.Published Online:01 Jan 1988https://doi.org/10.1152/physrev.1988.68.1.177MoreSectionsPDF (8 MB)Download PDF ToolsExport citationAdd to favoritesGet permissionsTrack citations ShareShare onFacebookTwitterLinkedInWeChat Previous Back to Top Next Download PDF FiguresReferencesRelatedInformation Cited ByEngineered Protein Model of the ATP synthase H+- Channel Shows No Salt Bridge at the Rotor-Stator Interface27 July 2018 | Scientific Reports, Vol. 8, No. 1Effects of essential oil decanal on growth and transcriptome of the postharvest fungal pathogen Penicillium expansumPostharvest Biology and Technology, Vol. 145A Multiscale Agent-Based Model for the Investigation of E. coli K12 Metabolic Response During Biofilm Formation14 September 2018 | Bulletin of Mathematical Biology, Vol. 7Stronger selective constraint on downstream genes in the oxidative phosphorylation pathway of cetaceans8 December 2017 | Journal of Evolutionary Biology, Vol. 31, No. 2Taking Electrons out of Bioelectronics: From Bioprotonic Transistors to Ion Channels14 March 2017 | Advanced Science, Vol. 4, No. 7Ectopic expression of the ATP synthase β subunit on the membrane of PC-3M cells supports its potential role in prostate cancer metastasis16 February 2017 | International Journal of Oncology, Vol. 50, No. 4Pulsatile shear stress increased mitochondrial membrane potential: Implication of Mn-SODBiochemical and Biophysical Research Communications, Vol. 388, No. 2Metabolism28 May 2008Hydrogen Bonds with Large Proton Polarizability and Proton Transfer Processes in Electrochemistry and Biology14 March 2007Rarity of Somatic Mutation and Frequency of Normal Sequence Variation Detected in Sporadic Colon Adenocarcinoma Using High-Throughput cDNA Sequencing17 September 2007 | Bioinformatics and Biology Insights, Vol. 1Structure and Mechanism of F 0 F 1 -Type ATP Synthases and ATPases22 November 2006Excessive ATP hydrolysis in ischemic myocardium by mitochondrial F1F0-ATPase: effect of selective pharmacological inhibition of mitochondrial ATPase hydrolase activityGary J. 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