生物
白色念珠菌
寄主(生物学)
免疫
微生物学
免疫系统
免疫学
生态学
标识
DOI:10.1016/j.chom.2024.08.005
摘要
During opportunistic pathogenic episodes, Candida albicans employs classical strategies such as the yeast-to-hyphae transition and immunogenic masking. In this issue of Cell Host & Microbe, Luo et al. unveil that the effector protein Cmi1 can be translocated into host cells and targets TBK1, thereby negatively regulating the host's antifungal immune responses.
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