特雷姆2
小胶质细胞
神经退行性变
生物
疾病
功能(生物学)
细胞
神经科学
阿尔茨海默病
受体
免疫学
内科学
细胞生物学
医学
遗传学
炎症
作者
Yongjin Yoo,Gernot Neumayer,Yohei Shibuya,Marius Marc-Daniel Mader,Marius Wernig
出处
期刊:Cell Stem Cell
[Elsevier]
日期:2023-10-01
卷期号:30 (10): 1392-1392
被引量:3
标识
DOI:10.1016/j.stem.2023.08.011
摘要
(Cell Stem Cell 30, 1043–1053.e1–e6; August 3, 2023) In the originally published version of this manuscript, the author Marius Marc-Daniel Mader was mistakenly listed with “Marc-Daniel Mader” as the surname, when in fact “Mader” is the correct surname of this author and “Marc-Daniel” is the middle name. This has now been corrected in the article online. A cell therapy approach to restore microglial Trem2 function in a mouse model of Alzheimer’s diseaseYoo et al.Cell Stem CellAugust 03, 2023In BriefMutations of the microglial receptor TREM2 leads to a premature neurodegeneration syndrome, and specific genetic variants are associated with Alzheimer’s disease. Yoo et al. show that high-efficiency microglia replacement represents a promising cell therapy to restore Trem2 function in a Trem2-dependent Alzheimer’s disease mouse model. Full-Text PDF
科研通智能强力驱动
Strongly Powered by AbleSci AI