先天性淋巴细胞
炎症
过敏性炎症
多巴胺
生物
哮喘
免疫学
先天免疫系统
神经科学
免疫系统
作者
Yingjiao Cao,Yu Li,Xiangyang Wang,Shaorui Liu,Yongmei Zhang,Gaoyu Liu,Shusen Ye,Yuhao Zheng,Jiacong Zhao,Xiaodong Zhu,Yingying Chen,Haixu Xu,Ding-Yun Feng,Dubo Chen,Ling Chen,Wangkai Liu,Wenjie Zhou,Zhi Zhang,Pan Zhou,Kai Deng
出处
期刊:Immunity
[Cell Press]
日期:2023-05-01
卷期号:56 (5): 1148-1151
被引量:3
标识
DOI:10.1016/j.immuni.2023.03.019
摘要
(Immunity 56, 320–335.e1–e9; February 14, 2023) The authors noticed that Figure 5D depicting IL-5+ ILC2 is a duplicate of Figure 5C depicting Ki-67+ ILC2. Figure 5D is incorrect and has been corrected. The corrected figure panel does not affect the conclusions. The authors apologize for any confusion that arose from the incorrect figure. Additionally, in original Figures S2F–S2L, the legend color for DA group was changed during file format conversion. The authors corrected this and made it consistent with the color used in other panels. The authors apologize for any inconvenience this caused. Dopamine inhibits group 2 innate lymphoid cell-driven allergic lung inflammation by dampening mitochondrial activityCao et al.ImmunityJanuary 23, 2023In BriefNeuroimmune interactions play an emerging role in the regulation of ILC2 and allergic inflammation, but the underlying mechanism remains to be fully understood. Cao et al. report that dopamine constrains ILC2 responses and alleviates allergic airway inflammation via DRD1 receptor. Full-Text PDF
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