医学
椎板成形术
后纵韧带骨化
骨化
外科
脊髓病
后纵韧带
后凸
减压
狭窄
再狭窄
脊髓
放射科
射线照相术
精神科
支架
作者
Masao Koda,Takeo Furuya,Akihiko Okawa,Satoshi Maki,Yasushi Ijima,Junya Saito,Mitsuhiro Kitamura,Sumihisa Orita,Kazuhide Inage,Seiji Ohtori,Tsuneyuki Abe,Hiroshi Noguchi,Toru Funayama,Kenji Suzuki,Hiroshi Kumagai,Katsuya Nagashima,Masashi Yamazaki
标识
DOI:10.1016/j.inat.2018.03.006
摘要
We report two cases of restenosis caused by the progression of thickness of ossification of the posterior longitudinal ligament (OPLL) seven and more years after laminoplasty, resulting in neurological deterioration needed for revision anterior decompressive surgeries. Neurological recovery after revision anterior excision of OPLL was poor. In both cases, the patients had progressive OPLL, with a non-ossified segment of the ossification foci, in common. After laminoplasty, they also both exhibited osseous fusion of the elevated laminae, but there was discontinuity at the interlaminar space at the peak level of OPLL. Discontinuity of the osseous fusion in the elevated laminae might cause mechanical stress increases at the non-ossified segment of the OPLL and could lead to the progression of OPLL. The present cases showed that long-term progression of OPLL can induce neurological deterioration even after sufficient posterior decompression by laminoplasty. Therefore, when considering risk factors that may be predictive of the progression of OPLL after laminoplasty, it is important to perform strict follow-up examination to check for progression to reduce the risk of myelopathy symptoms that are indicative of neurological deterioration.
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