医学
肝硬化
门脉高压
瞬态弹性成像
结节性再生增生
病因学
内科学
门静脉血栓形成
胃肠病学
肝细胞癌
放射科
肝纤维化
作者
Raj Vuppalanchi,Karan Mathur,Maximillian Pyko,Niharika Samala,Naga Chalasani
出处
期刊:Hepatology
[Lippincott Williams & Wilkins]
日期:2018-07-17
卷期号:68 (6): 2438-2440
被引量:35
摘要
Noncirrhotic portal hypertension (NCPH) is often a diagnostic challenge due to signs and symptoms of portal hypertension that overlap with cirrhosis. The etiology of NCPH is broadly classified as prehepatic, hepatic (presinusoidal and sinusoidal) and posthepatic.1 Some common etiologies of NCPH encountered in clinical practice include portal vein thrombosis (prehepatic) and nodular regenerative hyperplasia (hepatic). Liver histology, although considered gold standard to exclude cirrhosis in individuals with suspected NCPH, is often limited by subtle histologic features or inadequate sampling. Liver stiffness measurements (LSMs) by vibration‐controlled transient elastography may provide clinically important information to distinguish NCPH from cirrhosis by revealing normal LSM in prehepatic and presinusoidal NCPH.
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