Infection induces tissue-resident memory NK cells that safeguard tissue health

(Immunity 56, 531–546.e1–e6; March 14, 2023) Two typographical errors were introduced during the figure reformatting in the final copyediting process. In the key to Figure 2D, the pink segment of the bar graph should correspond to Eomes+CD49b+ cells. In the key to Figure 7D, the second line of the key should read “Tnfsf10−/− NKRM”, as was noted in the figure legend. The authors apologize for any confusion caused. Figure 2DNK cells are recruited and retained in the SG after MCMV infection (original)View Large Image Figure ViewerDownload Hi-res image Download (PPT)Figure 7DNKRM cells eliminate SG CD4+ T cells in a TRAIL-dependent manner to maintain SG function (corrected)View Large Image Figure ViewerDownload Hi-res image Download (PPT)Figure 7DNKRM cells eliminate SG CD4+ T cells in a TRAIL-dependent manner to maintain SG function (original)View Large Image Figure ViewerDownload Hi-res image Download (PPT) Infection induces tissue-resident memory NK cells that safeguard tissue healthSchuster et al.ImmunityFebruary 10, 2023In BriefImmune activation can be damaging if responses are not adequately controlled. Schuster et al. reveal that following cytomegalovirus infection, circulating natural killer cells are recruited into non-lymphoid tissues, including the salivary glands, where they establish a tissue-resident, memory-like population that prevents immune-mediated damage and safeguards tissue health by preventing autoimmunity. Full-Text PDF