NADPH氧化酶
生物
水通道蛋白
光合作用
效应器
植物
植物免疫
病菌
细胞生物学
活性氧
警卫室
拟南芥
生物化学
基因
微生物学
突变体
作者
Olivier Rodrigues,Libo Shan
标识
DOI:10.1016/j.tplants.2021.10.002
摘要
Stomatal movements are essential for plants to regulate photosynthesis rate, water status, and immunity. Upon stress stimulation, the production of hydrogen peroxide (H2O2) in the apoplasts and its accumulation within the guard cells are among key determinatives for stomatal closure. The regulatory mechanisms of H2O2 production and transport under plant–pathogen interaction and drought stress response in stomata are important fields of research. Specifically, the regulation of NADPH oxidases and aquaporins appears to be crucial in H2O2-controlled stomatal closure. In this review, we summarize how the calcium-dependent and calcium-independent mechanisms activate RESPIRATORY BURST OXIDASE HOMOLOG (RBOH)D/F NADPH oxidases and the aquaporin PIP2;1 to induce stomatal closure, and highlight how the H2O2 production is targeted by pathogen toxins and effectors to counteract plant immunity.
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