神经炎症
疾病
阿尔茨海默病
医学
计算生物学
小胶质细胞
生物信息学
神经科学
生物
免疫学
炎症
病理
作者
Zhixin Xie,Linxi Li,Weizhong Hou,Zhongxi Fan,Li‐Fan Zeng,Limin He,Yunxiang Ji,Jingbai Zhang,F J Wang,Xing Zhou,Yezhong Wang,Yongyi Ye
标识
DOI:10.1186/s12967-025-06112-2
摘要
We demonstrated that Oas1g was identified as a hub ISG in AD and can downregulate the activation of IFN-β and STAT1, reducing the expression of ISGs in neuroinflammation. Oas1g might potentially be a beneficial candidate for both prevention and treatment of AD.
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