自噬
异甘草素
医学
安普克
PI3K/AKT/mTOR通路
ULK1
血管病学
依瓦布拉定
内科学
药理学
信号转导
蛋白激酶A
激酶
细胞生物学
细胞凋亡
生物
心率
生物化学
血压
作者
Liying Shen,Zhu YingWei,Zhenfeng Chen,Feng Shen,Weiwei Yu,L Zhang
标识
DOI：10.1186/s12872-024-04054-z
摘要
Ischemia reperfusion (IR) causes impaired myocardial function, and autophagy activation ameliorates myocardial IR injury. Isoliquiritigenin (ISO) has been found to protect myocardial tissues via AMPK, with exerting anti-tumor property through autophagy activation. This study aims to investigate ISO capacity to attenuate myocardial IR through autophagy activation mediated by AMPK/mTOR/ULK1 signaling.
科研通智能强力驱动
Strongly Powered by AbleSci AI
Polymer72
无名老大
乐乐乐乐乐乐
VDC
qq1083716237
模糊中正
爱静静
Yangyang
y915840635
美满疾
cnspower
Hayat
andrele
加菲丰丰
wax
萧水白
传奇3
不安青牛
毕不了业要赔钱
pcr163
皖公网安备34019202002308
