癌症研究
黑色素瘤
降级(电信)
生物
激酶
极光A激酶
细胞生物学
泛素
化学
医学
计算机科学
基因
电信
生物化学
作者
Wei Sun,Yongjia Jin,Chuanyuan Wei,Yu Xu,Wanlin Liu,Jingqin Zhong,Zijian Zou,Xinyi Lin,Yang Xiang,Yong Chen
标识
DOI:10.1016/j.ejca.2023.04.005
摘要
CDCA2, which was upregulated in melanoma, enhanced AURKA protein stability by inhibiting SMAD specific E3 ubiquitin protein ligase 1-mediated AURKA ubiquitination, thus playing a carcinogenic role in melanoma progression.
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