SMYD3 activates the TCA cycle to promote M1-M2 conversion in macrophages

H3K4me3 组蛋白H3 生物 巨噬细胞极化 组蛋白 细胞生物学 丙酮酸脱氢酶复合物 组蛋白甲基化 生物化学 基因表达 表型 基因 DNA甲基化 发起人
作者
Wenqiang Zhu,Lina Liu,Jinjing Wu,Renzhuo Gao,Liying Fu,Yang Xiao-hong,Yang Zou,Shu‐Hua Zhang,Daya Luo
出处
期刊:International Immunopharmacology [Elsevier]
卷期号:127: 111329-111329 被引量:3
标识
DOI:10.1016/j.intimp.2023.111329
摘要

SMYD3 refers to a histone lysine methyltransferase from the SMYD family, which acts as a gene transcriptional regulator chiefly through catalysis of the histone subunit 3 at lysine 4 trimethylation (H3K4me3). Great progress has been made that epigenetic modification plays a pivotal role in regulating macrophage polarization. However, the effects of the histone lysine methyltransferase SMYD3 on macrophage polarization and phenotypic switching are unclear. We found that LPS/IFN-γ-stimulated macrophages gradually transformed from M1 to M2 in the late stage, and SMYD3 played a key role in this process. As demonstrated by RNA-seq assessment, SMYD3 prominently activated a metabolic pathway known as TCA cycle inside macrophages during M1-M2 conversion. Besides, by modifying H3K4me3 histone, the target genes regulated by SMYD3 were identified via the ChIP-seq assessment, including citrate synthase (CS), succinate dehydrogenase complex subunit C (SDHC) and pyruvate carboxylase (PC). SMYD3 activated the transcriptional activities of the metabolic enzymes CS, SDHC and PC through H3K4me3 by causing the aggregation of citrate, an intramacrophage metabolite, and the depletion of succinate. And additionally, it facilitated the generation of ROS, as well as the expressions of genes associated with mitochondrial respiratory chain complexes. This increased ROS production ultimately induced mitophagy, triggering the M1 to M2 phenotype switch in the macrophages. Our study provides a detailed intrinsic mechanism in the macrophage phenotypic transition process, in short, SMYD3 promotes the M1-M2 conversion of macrophages by activating the TCA cycle through the simultaneous regulation of the transcriptional activities of the metabolic enzymes CS, SDHC and PC.
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