ATF3
血管平滑肌
血管紧张素II
激活转录因子
生物
基因敲除
癌症研究
未折叠蛋白反应
内分泌学
免疫学
细胞凋亡
细胞生物学
基因表达
内质网
发起人
基因
生物化学
平滑肌
血压
作者
Ying Wen,Yingying Liu,Qiang Li,Jin‐Lin Tan,Xing Fu,Yiwen Liang,Yonghua Tuo,Luhao Liu,Xueqiong Zhou,Dongkai LiuFu,Xuejiao Fan,Chaofei Chen,Zheng Chen,Zhouping Wang,Shunyang Fan,Renjing Liu,Lei Pan,Y.-Q. Zhang,Wai Ho Tang
标识
DOI:10.1161/circresaha.124.324323
摘要
Abdominal aortic aneurysm (AAA) is a catastrophic disease with little effective therapy, likely due to the limited understanding of the mechanisms underlying AAA development and progression. ATF3 (activating transcription factor 3) has been increasingly recognized as a key regulator of cardiovascular diseases. However, the role of ATF3 in AAA development and progression remains elusive.
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