自噬
体外
p38丝裂原活化蛋白激酶
小RNA
医学
细胞生物学
化学
癌症研究
药理学
生物
细胞凋亡
信号转导
基因
生物化学
MAPK/ERK通路
作者
Ying Chen,Liyong Huang,Lei Wang,Lingyun Chen,Wenhua Ren,Wenke Zhou
出处
期刊:Food & Function
[The Royal Society of Chemistry]
日期:2017-01-01
卷期号:8 (12): 4675-4683
被引量:10
摘要
(1) EGCG prevented miRNA dysregulation after SAH; (2) multi-target mechanisms of EGCG might rely on its regulatory roles on miRNAs expression, such as those miRNAs targeting p38, Ca2+, and autophagic activation; (3) the differential expression of miRNAs might determine the therapeutic efficacy of different concentration of EGCG.
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