坏死性下垂
蛋白尿
足细胞
医学
肾损伤
内科学
肾
心脏病学
生物
蛋白尿
细胞凋亡
生物化学
程序性细胞死亡
作者
Jeong Suk Kang,Namjun Cho,Seong Woo Lee,Jeong Geon Lee,Ji‐Hye Lee,Jawoon Yi,Min Sun Choi,Samel Park,Hyo‐Wook Gil,Joon Cheol Oh,Seung Seob Son,Mi Ju Park,Jong‐Seok Moon,Donghyeong Lee,So-Young Kim,Seung-Hoon Yang,Sang Soo Kim,Eun Soo Lee,Choon Hee Chung,Jihwan Park,Eun Young Lee
标识
DOI：10.1016/j.metabol.2024.155982
摘要
Receptor-interacting protein kinase (RIPK)3 is an essential molecule for necroptosis and its role in kidney fibrosis has been investigated using various kidney injury models. However, the relevance and the underlying mechanisms of RIPK3 to podocyte injury in albuminuric diabetic kidney disease (DKD) remain unclear. Here, we investigated the role of RIPK3 in glomerular injury of DKD.
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