蛋白激酶B
PI3K/AKT/mTOR通路
癌症研究
棕榈酰化
脂肪性肝炎
脂肪肝
癌变
化学
生物
细胞生物学
信号转导
生物化学
内科学
医学
疾病
半胱氨酸
基因
酶
作者
Lang Bu,Zhengkun Zhang,Jianwen Chen,Yanli Fan,Jin‐He Guo,Yaqing Su,Chunlin Wang,Xiaomei Zhang,Xueji Wu,Qiwei Jiang,Bing Gao,Lei Wang,Kunpeng Hu,Xiang Zhang,Wei Xie,Wenyi Wei,Ming Kuang,Jianping Guo
出处
期刊:Gut
[BMJ]
日期:2024-01-08
卷期号:73 (7): 1156-1168
被引量:5
标识
DOI:10.1136/gutjnl-2023-330826
摘要
Whether and how the PI3K-AKT pathway, a central node of metabolic homeostasis, is responsible for high-fat-induced non-alcoholic steatohepatitis (NASH) and hepatocellular carcinoma (HCC) remain a mystery. Characterisation of AKT regulation in this setting will provide new strategies to combat HCC.
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