DNA损伤
DNA修复
同源重组
非同源性末端接合
体细胞
生物
细胞生物学
DNA
DNA损伤修复
DNA修复蛋白XRCC4
癌症研究
DNA错配修复
遗传学
基因
作者
Wil L. Santivasi,Fen Xia
标识
DOI:10.1089/ars.2013.5668
摘要
By utilizing the strategies outlined above, it may be possible to exploit differences between tumor and somatic cell DRR pathways, specifically their DSB repair mechanisms, to improve the therapeutic ratio of IR.
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