医学
超细粒子
呼吸系统
北京
环境卫生
二氧化氮
毒理
儿科
内科学
气象学
化学
生物
有机化学
中国
法学
物理
政治学
作者
Jiakun Fang,Xiaoming Song,Hongbing Xu,Rongshan Wu,Jing Song,Yunfei Xie,Xin Xu,Yueping Zeng,Tong Wang,Yutong Zhu,Ningman Yuan,Jinzhu Jia,Baoping Xu,Wei Huang
出处
期刊:Thorax
[BMJ]
日期:2021-07-22
卷期号:77 (4): 391-397
被引量:23
标识
DOI:10.1136/thoraxjnl-2021-217017
摘要
Background Ambient fine particulate matter with aerodynamic diameter less than 2.5 µm (PM 2.5 ) has been associated with deteriorated respiratory health, but evidence on particles in smaller sizes and childhood respiratory health has been limited. Methods We collected time-series data on daily respiratory emergency room visits (ERVs) among children under 14 years old in Beijing, China, during 2015–2017. Concurrently, size-fractioned number concentrations of particles in size ranges of 5–560 nm (PNC 5–560 ) and mass concentrations of PM 2.5 , black carbon (BC) and nitrogen dioxide (NO 2 ) were measured from a fixed-location monitoring station in the urban area of Beijing. Confounder-adjusted Poisson regression models were used to estimate excessive risks (ERs) of particle size fractions on childhood respiratory ERVs, and positive matrix factorisation models were applied to apportion the sources of PNC 5–560 . Results Among the 136 925 cases of all-respiratory ERVs, increased risks were associated with IQR increases in PNC 25–100 (ER=5.4%, 95% CI 2.4% to 8.6%), PNC 100–560 (4.9%, 95% CI 2.5% to 7.3%) and PM 2.5 (1.3%, 95% CI 0.1% to 2.5%) at current and 1 prior days (lag0–1). Major sources of PNC 5–560 were identified, including nucleation (36.5%), gasoline vehicle emissions (27.9%), diesel vehicle emissions (18.9%) and secondary aerosols (10.6%). Emissions from gasoline and diesel vehicles were found of significant associations with all-respiratory ERVs, with increased ERs of 6.0% (95% CI 2.5% to 9.7%) and 4.4% (95% CI 1.7% to 7.1%) at lag0–1 days, respectively. Exposures to other traffic-related pollutants (BC and NO 2 ) were also associated with increased respiratory ERVs. Conclusion Our findings suggest that exposures to higher levels of PNC 5–560 from traffic emissions could be attributed to increased childhood respiratory morbidity, which supports traffic emission control priority in urban areas.
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