NAD+激酶
大肠杆菌
生物
生物化学
神经退行性变
氧化还原
氧化应激
线粒体
新陈代谢
氧化磷酸化
转录组
细胞生物学
化学
基因
酶
基因表达
医学
疾病
有机化学
病理
作者
Xingxiu Pan,Mina L. Heacock,Evana N. Abdulaziz,Sara Violante,Austin L. Zuckerman,Nirajan Shrestha,Canglin Yao,Russell P. Goodman,Justin R. Cross,Valentin Cracan
标识
DOI:10.1038/s41589-023-01460-w
摘要
Impaired redox metabolism is a key contributor to the etiology of many diseases, including primary mitochondrial disorders, cancer, neurodegeneration and aging. However, mechanistic studies of redox imbalance remain challenging due to limited strategies that can perturb redox metabolism in various cellular or organismal backgrounds. Most studies involving impaired redox metabolism have focused on oxidative stress; consequently, less is known about the settings where there is an overabundance of NADH reducing equivalents, termed reductive stress. Here we introduce a soluble transhydrogenase from Escherichia coli (EcSTH) as a novel genetically encoded tool to promote reductive stress in living cells. When expressed in mammalian cells, EcSTH, and a mitochondrially targeted version (mitoEcSTH), robustly elevated the NADH/NAD+ ratio in a compartment-specific manner. Using this tool, we determined that metabolic and transcriptomic signatures of the NADH reductive stress are cellular background specific. Collectively, our novel genetically encoded tool represents an orthogonal strategy to promote reductive stress. A soluble bacterial transhydrogenase from Escherichia coli (EcSTH) was validated as a genetically encoded tool to induce NADH reductive stress in mammalian cells revealing unique transcriptional and metabolic signatures.
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