伊诺斯
血管内皮生长因子受体
蛋白激酶B
缺血
PI3K/AKT/mTOR通路
信号转导
医学
动脉发生
内科学
内分泌学
细胞生物学
生物
一氧化氮
一氧化氮合酶
作者
Lu Cao,W. Ding,Ya-Shuo Feng,Chong Guan,Li Liu,Hongyu Xie,Kewei Yu,Dongyan Xu,Lijuan Zhao,Xuan Sha,Xiaoman Deng,Shixiu Wu,Yangrui Wang,Yi Wu,Tingting Zhang,Nianhong Wang
标识
DOI:10.1016/j.brainresbull.2025.111192
摘要
EA upregulated irisin expression, reaching a peak after 7 days of EA and then stabilizing. EA facilitated vascular remodeling after cerebral ischemia, and this might be associated with the activation of the irisin-mediated VEGF/Akt/eNOS signaling pathway.
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