Integrated Omics Reveal the Molecular Characterization and Pathogenic Mechanism of Rosacea

蛋白质组 酒渣鼻 转录组 S100A9型 蛋白质组学 银屑病 生物 计算生物学 炎症 医学 免疫学 生物信息学 遗传学 基因 基因表达 痤疮
作者
Yiya Zhang,Yingxue Huang,Ben Wang,Wei Shi,Xi‐min Hu,Yaling Wang,Yi Guo,Hongfu Xie,Wenqin Xiao,Ji Li
出处
期刊:Journal of Investigative Dermatology [Elsevier]
卷期号:144 (1): 33-42.e2 被引量:8
标识
DOI:10.1016/j.jid.2023.05.028
摘要

Recent efforts have described the transcriptomic landscape of rosacea. However, little is known about its proteomic characteristics. In this study, the proteome and phosphoproteome of lesional skin, paired nonlesional skin, and healthy skin were analyzed by liquid chromatography coupled with tandem mass spectrometry. The molecular characteristics and potential pathogenic mechanism of rosacea were demonstrated by integrating the proteome, phosphoproteome, and previous transcriptome. The proteomic data revealed a significant upregulation of inflammation- and axon extension–related proteins in lesional skin and nonlesional skin versus in healthy skin, implying an inflammatory and nerve-hypersensitive microenvironment in rosacea skin. Of these, axon-related proteins (DPYSL2 and DBNL) were correlated with the Clinician's Erythema Assessment score, and neutrophil-related proteins (ELANE and S100A family) were correlated with the Investigator's Global Assessment score. Moreover, comorbidity-related proteins were differentially expressed in rosacea; of these, SNCA was positively correlated with Clinician's Erythema Assessment score, implying a potential correlation between rosacea and comorbidities. Subsequently, the integrated proteome and transcriptome demonstrated consistent immune disturbances at both the transcriptional and protein levels. The integrative analysis of the proteome and phosphoproteome revealed the key transcription factor network and kinase network that drive the dysregulation of immunity and vasculature in rosacea. In conclusion, our multiomics analysis enables more comprehensive insight into rosacea and offers an opportunity for, to our knowledge, previously unreported treatment strategies.
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