加压器
菊花
转录因子
热冲击系数
抑制因子
组蛋白
生物
抄写(语言学)
MYB公司
热休克蛋白
乙酰化
细胞生物学
热休克蛋白70
分子生物学
遗传学
植物
基因
语言学
哲学
作者
X.F. Wang,Y. Wang,Yuhan Jiang,Han Wang,Lijie Zhou,Fei Li,Likai Wang,Jiafu Jiang,Fadi Chen,Sumei Chen
标识
DOI:10.1093/plphys/kiae238
摘要
Abstract Excessive soil salinity not only hampers plant growth and development but can also lead to plant death. Previously, we found that heat shock factor A4 (CmHSFA4) enhances the tolerance of chrysanthemum (Chrysanthemum morifolium) to salt. However, the underlying molecular mechanism remains unclear. In this study, we identified a candidate MYB transcription factor, CmMYB121, which responded to salt stress. We observed that the CmMYB121 transcription is suppressed by CmHSFA4. Moreover, overexpression of CmMYB121 exacerbated chrysanthemum sensitivity to salt stress. CmHSFA4 directly bound to the promoter of CmMYB121 at the heat shock element (HSE). Protein–protein interaction assays identified an interaction between CmHSFA4 and CmMYBS3, a transcriptional repressor, and recruited the corepressor TOPLESS (CmTPL) to inhibit CmMYB121 transcription by impairing the H3 and H4 histone acetylation levels of CmMYB121. Our study demonstrated that a CmHSFA4-CmMYBS3-CmTPL complex modulates CmMYB121 expression, consequently regulating the tolerance of chrysanthemum to salt. The findings shed light on the responses of plants to salt stress.
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