炎症体
生长素
内质网
糖尿病性心肌病
串扰
上睑下垂
内科学
内分泌学
氧化应激
未折叠蛋白反应
炎症
PI3K/AKT/mTOR通路
内生
医学
产矿性
心肌病
细胞生物学
信号转导
生物
心力衰竭
激素
神经肽Y受体
受体
神经肽
物理
光学
作者
Fan Wang,Jingzhi Wang,Xin-Fang Liang,Zhenqiang Wu,Jing Xue,Liangyu Yin,Wen‐Ter Lai,Xiaohui Zhang
标识
DOI:10.1080/1061186x.2023.2295268
摘要
Aims Endoplasmic reticulum stress(ERS) can induce inflammation mediated by NLRP3 inflammatory bodies and link inflammation with oxidative stress in myocardial tissue. Ghrelin is an endogenous growth hormone-releasing peptide that has been proven to have multiple effects, such as regulating energy metabolism and inhibiting inflammation. However, the role of ghrelin in myocardial injury in diabetic rats and the mechanism have not been reported.
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