肿瘤微环境
癌症研究
克里唑蒂尼
肺癌
免疫系统
肿瘤进展
医学
免疫疗法
佐剂
细胞毒性T细胞
免疫学
癌症
化学
肿瘤科
内科学
体外
恶性胸腔积液
生物化学
作者
Natalie K. Horvat,Sara Chocarro,Oriana Marques,Tobias Bauer,Ruiyue Qiu,Alberto Diaz-Jimenez,Barbara Helm,Yuanyuan Chen,Stefan Sawall,Richard Sparla,Lu Su,Ursula Klingmüller,Matthias Barz,Matthias W. Hentze,Rocı́o Sotillo,Martina U. Muckenthaler
出处
期刊:ACS Nano
[American Chemical Society]
日期:2024-04-16
卷期号:18 (17): 11025-11041
被引量:6
标识
DOI:10.1021/acsnano.3c08335
摘要
ALK-positive NSCLC patients demonstrate initial responses to ALK tyrosine kinase inhibitor (TKI) treatments, but eventually develop resistance, causing rapid tumor relapse and poor survival rates. Growing evidence suggests that the combination of drug and immune therapies greatly improves patient survival; however, due to the low immunogenicity of the tumors, ALK-positive patients do not respond to currently available immunotherapies. Tumor-associated macrophages (TAMs) play a crucial role in facilitating lung cancer growth by suppressing tumoricidal immune activation and absorbing chemotherapeutics. However, they can also be programmed toward a pro-inflammatory tumor suppressive phenotype, which represents a highly active area of therapy development. Iron loading of TAMs can achieve such reprogramming correlating with an improved prognosis in lung cancer patients. We previously showed that superparamagnetic iron oxide nanoparticles containing core-cross-linked polymer micelles (SPION-CCPMs) target macrophages and stimulate pro-inflammatory activation. Here, we show that SPION-CCPMs stimulate TAMs to secrete reactive nitrogen species and cytokines that exert tumoricidal activity. We further show that SPION-CCPMs reshape the immunosuppressive
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