PI3K/AKT/mTOR通路
蛋白激酶B
细胞凋亡
炎症
体内
缺血
医学
再灌注损伤
药理学
LY294002型
化学
内科学
生物
生物化学
生物技术
作者
Yuanping Wang,J.X. Zheng,X. Xiao,Cailing Feng,Yinghong Li,Hui Su,Ding Yuan,Q. Wang,Pinyuan Huang,L. W. Jin
标识
DOI:10.1142/s0192415x24500186
摘要
against myocardial I/R injury was primarily dependent on PI3K/Akt pathway activation to inhibit inflammation and cardiomyocyte apoptosis. This study provides new evidence for the use of GRd as a cardiovascular drug.
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