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SETDB1 promotes gastric carcinogenesis and metastasis via upregulation of CCND1 and MMP9 expression

下调和上调 癌症研究 癌变 细胞周期蛋白D1 转移 转录因子 MMP9公司 化学 分子生物学 生物 癌症 基因 遗传学 细胞周期
作者
Wenjing Shang,Yue Wang,Xiuming Liang,Tongyu Li,Wei Shao,Fen Liu,Xiujie Cui,Yuanyuan Wang,Lin Lv,Li Chai,Lingxin Qu,Lixin Zheng,Jihui Jia
标识
DOI:10.1002/path.5568
摘要

SETDB1 is a histone lysine methyltransferase that has critical roles in cancers. However, its potential role in gastric cancer (GC) remains obscure. Here, we mainly investigate the clinical significance and the possible role of SETDB1 in GC. We find that SETDB1 expression is upregulated in GC tissues and its high-level expression was a predictor of poor prognosis in patients. Overexpression of SETDB1 promoted cell proliferation and metastasis, while SETDB1 suppression had an opposite effect both in vitro and in vivo. Mechanistically, SETDB1 was shown to interact with ERG to promote the transcription of cyclin D1 (CCND1) and matrix metalloproteinase 9 (MMP9) through binding to their promoter regions. In addition, the expression of SETDB1 was also enhanced by the transcription factor TCF4 at the transcriptional level in GC. Furthermore, SETDB1 expression was found to be induced by Helicobacter pylori (H. pylori) infection in a TCF4-dependent manner. Taken together, our results indicate that SETDB1 is aberrantly overexpressed in GC and plays key roles in gastric carcinogenesis and metastasis via upregulation of CCND1 and MMP9. Our work also suggests that SETDB1 could be a potential oncogenic factor and a therapeutic target for GC. © 2020 The Pathological Society of Great Britain and Ireland. Published by John Wiley & Sons, Ltd.
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