胸腺基质淋巴细胞生成素
白细胞介素33
免疫学
呼吸上皮
先天性淋巴细胞
炎症
过敏性炎症
细胞因子
上皮
肺
过继性细胞移植
白细胞介素13
医学
免疫系统
白细胞介素
获得性免疫系统
病理
T细胞
内科学
作者
Pauline Schmitt,Anais Dufros Duval,Matthieu Camus,Emma Lefrançais,Stéphane Roga,Cécile Dedieu,Nathalie Ortéga,Elisabeth Bellard,Emilie Mirey,Emmanuelle Mouton-Barbosa,Odile Burlet‐Schiltz,Anne Gonzalez de Peredo,Corinne Cayrol,Jean‐Philippe Girard
摘要
Epithelium-derived cytokines or alarmins, such as interleukin-33 (IL-33) and thymic stromal lymphopoietin (TSLP), are major players in type 2 immunity and asthma. Here, we demonstrate that TNF-like ligand 1A (TL1A) is an epithelial alarmin, constitutively expressed in alveolar epithelium at steady state in both mice and humans, which cooperates with IL-33 for early induction of IL-9high ILC2s during the initiation of allergic airway inflammation. Upon synergistic activation by IL-33 and TL1A, lung ILC2s acquire a transient IL-9highGATA3low "ILC9" phenotype and produce prodigious amounts of IL-9. A combination of large-scale proteomic analyses, lung intravital microscopy, and adoptive transfer of ILC9 cells revealed that high IL-9 expression distinguishes a multicytokine-producing state-of-activated ILC2s with an increased capacity to initiate IL-5-dependent allergic airway inflammation. Similar to IL-33 and TSLP, TL1A is expressed in airway basal cells in healthy and asthmatic human lungs. Together, these results indicate that TL1A is an epithelium-derived cytokine and an important cofactor of IL-33 in the airways.
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