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Exploring macrophage heterogeneity in IgA nephropathy: Mechanisms of renal impairment and current therapeutic targets
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已完结
Intensity of Macrophage Infiltration in Glomeruli Predicts Response to Immunosuppressive Therapy in Patients with IgA Nephropathy
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Glomerular expression and urinary excretion of fatty acid-binding protein 4 in IgA nephropathy
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Intensity of Macrophage Infiltration in Glomeruli Predicts Response to Immunosuppressive Therapy in Patients with IgA Nephropathy
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Crucial Role of AIM/CD5L in the Development of Glomerular Inflammation in IgA Nephropathy
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SPAK plays a pathogenic role in IgA nephropathy through the activation of NF-κB/MAPKs signaling pathway
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Targeting APRIL in the treatment of glomerular diseases
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Immune checkpoint activity exacerbate renal interstitial fibrosis progression by enhancing PD-L1 expression in renal tubular epithelial cells
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Exosomal CCL2 from Tubular Epithelial Cells Is Critical for Albumin-Induced Tubulointerstitial Inflammation
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Selective tubular activation of hypoxia-inducible factor-2α has dual effects on renal fibrosis
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