串扰
腺泡细胞
下调和上调
2型糖尿病
小岛
胰腺
电池类型
细胞
胰岛
细胞生物学
糖尿病
内分泌学
生物
内科学
医学
生物化学
物理
光学
基因
作者
Shirin Geravandi,Amin Ardestani
标识
DOI:10.1016/j.tem.2023.08.006
摘要
Cellular crosstalk plays a vital role in maintaining pancreas homeostasis. Recently, in Cell Metabolism, Basile et al. demonstrated that aberrant upregulation of acinar-cell-specific pancreatic elastase CELA3B within endocrine islets reduces β-cell viability in type 2 diabetes (T2D). This identifies detrimental acinar–β-cell crosstalk as a novel pathogenic mechanism in diabetes.
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