克拉斯
转录因子
癌症研究
突变体
生物
突变
计算生物学
遗传学
基因
作者
Connor A. Ott,Andrew E. Aplin
标识
DOI：10.1016/j.trecan.2023.07.016
摘要
Recent advances in targeting mutant KRAS are limited by resistance. A recent study in Nature Cancer by Hagenbeek et al. utilizes a novel inhibitor that targets the TEAD transcription factor, GNE-7883, to overcome resistance to KRAS inhibitors. Thus, TEAD inhibitors may maximize the durability of KRAS inhibitors in patients with cancer.
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Estrella
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Singularity
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