PTEN公司
心肌纤维化
纤维化
医学
心力衰竭
病态的
射血分数保留的心力衰竭
蛋白激酶B
PI3K/AKT/mTOR通路
一氧化氮合酶
内科学
一氧化氮
信号转导
心脏病学
射血分数
生物
细胞生物学
作者
Hongjun You,Qiling Gou,Mengya Dong,Feng-Jun Chang,Jiancheng Xiu
标识
DOI:10.1016/j.bbrc.2024.150589
摘要
Heart failure with preserved ejection fraction (HFpEF) is a challenging condition to treat with myocardial fibrosis being a pivotal pathological component. Previous studies have suggested a role for inducible nitric oxide synthase (iNOS) in the progression of this condition, but the precise mechanisms remain unclear. This study aimed to investigate the role of iNOS in HFpEF-related myocardial fibrosis and identify potential therapeutic targets.
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