动静脉畸形
医学
单核细胞
病理
炎症
免疫系统
血管生成
发病机制
小胶质细胞
先天免疫系统
癌症研究
免疫学
放射科
作者
Li Ma,Xiaonan Zhu,Chaoliang Tang,Peipei Pan,Alka Yadav,Rich Liang,Kelly Press,Jeffrey Nelson,Hua Su
标识
DOI:10.1177/0271678x241236008
摘要
Myeloid immune cells are abundant in both ruptured and unruptured brain arteriovenous malformations (bAVMs). The role of central nervous system (CNS) resident and circulating monocyte-derived macrophages in bAVM pathogenesis has not been fully understood. We hypothesize that CNS resident macrophages enhance bAVM development and hemorrhage. RNA sequencing using cultured endothelial cells (ECs) and mouse bAVM samples revealed that downregulation of two bAVM causative genes, activin-like kinase 1
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