去骨瓣减压术
颅骨成形术
医学
外科
减压
脑疝
颅内压
创伤性脑损伤
精神科
颅骨
作者
Gopalakrishnan M. Sasidharan,Nagesh C. Shanbhag,Dhaval Shukla,Subhas Konar,Dhananjaya I. Bhat,Bhagavatula Indira Devi
标识
DOI:10.3389/fneur.2018.00977
摘要
Decompressive craniectomy has become the definitive surgical procedure to manage medically intractable rise in intracranial pressure due to stroke and traumatic brain injury. With incoming evidence from recent multi-centric randomized controlled trials to support its use, we could expect a significant rise in the number of patients who undergo this procedure. Although one would argue that the procedure reduces mortality only at the expense of increasing the proportion of the severely disabled, what is not contested is that patients face the risk of a large number of complications after the operation and that can further compromise the quality of life. Decompressive craniectomy, which is designed to overcome the space constraints of the Monro Kellie doctrine, perturbs the cerebral blood and CSF flow dynamics. Resultant complications occur days to months after the surgical procedure in a time pattern that can be anticipated with advantage in managing them. New or expanding hematomas that occur within the first few days can be life-threatening and we recommend CT scans at 24 and 48 hours postoperatively to detect them. Surgeons should also be mindful of the myriad manifestations of peculiar complications like the syndrome of the trephined and neurological deterioration due to paradoxical herniation which may occur many months after the decompression. A sufficiently large frontotemporoparietal craniectomy, 15 cm in diameter, increases the effectiveness of the procedure and reduces chances of external cerebral herniation. An early cranioplasty, as soon as the brain is lax, appears to be a reasonable choice to mitigate many of the late complications. Complications, their causes, consequences and measures to manage them are described in this chapter.
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