生物
髓系白血病
癌症研究
白血病
髓样
干细胞
细胞生长
遗传增强
细胞
免疫学
作者
Malini Gupta,Britta Will
标识
DOI：10.1016/j.stem.2022.02.007
摘要
Adaptive aberrant gene regulation is a hallmark of malignant growth and therapy resistance in acute myeloid leukemia (AML). In this issue of Cell Stem Cell, Eagle et al. identified oncogenic enhancer-driven overexpression of selenophosphate synthetase 2 (SEPHS2) as an opportunity for targeted mitigation of malignant cell growth in AML.
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