SIRT3
医学
神经保护
缺血
线粒体
线粒体融合
内分泌学
线粒体生物发生
下调和上调
内科学
细胞生物学
麻醉
锡尔图因
生物
生物化学
线粒体DNA
乙酰化
基因
作者
Hongbin Chen,Ji Liu,Manli Chen,Zengyu Wei,Jinjin Yuan,Wenwen Wu,Zhiyun Wu,Zhijian Zheng,Zijun Zhao,Qiang Lin,Nan Liu
标识
DOI:10.1016/j.clnu.2024.06.001
摘要
Optical atrophy 1 (OPA1), a protein accountable for mitochondrial fusion, facilitates the restoration of mitochondrial structure and function following cerebral ischemia/reperfusion (I/R) injury. The OPA1-conferred mitochondrial protection involves its expression and activity, which can be improved by SIRT3 in non-cerebral ischemia. Nevertheless, it remains obscure whether SIRT3 enhances the expression and activity of OPA1 after cerebral I/R injury.
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