生物
结直肠癌
肠道菌群
代谢组
慢性应激
大肠癌小鼠模型的建立
癌症研究
癌症
免疫学
内科学
代谢物
内分泌学
医学
遗传学
作者
Qiuhua Cao,Mingrui Zhao,Yali Su,Siliang Liu,Yanting Lin,Huijuan Da,Chongxiu Yue,Yiming Liu,Dongquan Jing,Qixiang Zhao,Ning Liu,Juan Du,Zhanjie Zuo,Yao Fu,Anqi Chen,Lutz Birnbaumer,Yong Yang,Beiying Dai,Xinghua Gao
出处
期刊:Cancer Research
[American Association for Cancer Research]
日期:2024-01-08
卷期号:84 (5): 771-784
被引量:28
标识
DOI:10.1158/0008-5472.can-22-3705
摘要
Chronic stress stimulates cancer stemness by reducing the intestinal abundance of L. johnsonii and its metabolite PCA to enhance β-catenin signaling, forming a basis for potential strategies to circumvent stress-induced cancer aggressiveness. See related commentary by McCollum and Shah, p. 645.
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