医学
心房颤动
自主神经系统
传出的
心脏病学
肺静脉
内科学
机制(生物学)
传入的
神经科学
心率
血压
心理学
认识论
哲学
作者
Varun Malik,Dominik Linz,Prashanthan Sanders
标识
DOI:10.1016/j.ccep.2023.08.003
摘要
Several complex mechanisms, working alone, or together, initiate and maintain atrial fibrillation (AF). At disease onset, pulmonary vein-atrial triggers, producing ectopy, predominate. Then, as AF progresses, a shift toward substrate occurs, which AF also self-perpetuates. The autonomic nervous system (ANS) plays an important role as trigger and substrate. Although the efferent arm of the ANS as AF trigger is well-established, there is emerging evidence to show that (1) the ANS is a substrate for AF and (2) afferent or regulatory ANS dysfunction occurs in AF patients. These findings could represent a mechanism for the progression of AF.
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