医学
中性粒细胞胞外陷阱
血管性血友病因子
发病机制
细胞外
心力衰竭
心脏病学
机制（生物学）
内科学
免疫学
炎症
血小板
细胞生物学
生物
哲学
认识论
作者
Ge Mang,Jianfeng Chen,Ping Sun,Ruishuang Ma,Jingwen Du,Xiaoqi Wang,Jingxuan Cui,Mian Yang,Zhonghua Tong,Xiangyu Yan,Dongni Wang,Huiqi Xie,Yujia Chen,Qiannan Yang,Yingjin Kong,Jiaqi Jin,Zhenhua Wu,Maomao Zhang,Bo Yu
标识
DOI：10.1093/eurheartj/ehae517
摘要
Heart failure (HF) is a leading cause of mortality worldwide and characterized by significant co-morbidities and dismal prognosis. Neutrophil extracellular traps (NETs) aggravate inflammation in various cardiovascular diseases; however, their function and mechanism of action in HF pathogenesis remain underexplored. This study aimed to investigate the involvement of a novel VWF-SLC44A2-NET axis in HF progression.
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