成纤维细胞活化蛋白
间充质干细胞
胶质瘤
间质细胞
癌症研究
CXCR4型
基质
生物
免疫组织化学
医学
病理
癌症
免疫学
趋化因子
内科学
免疫系统
作者
Petr Bušek,Eva Balážiová,Ivana Matrasová,Marek Hilser,Robert Tomáš,Martin Syrůček,Zuzana Zemanová,E Křepela,Jaromír Běláček,Aleksi Šedo
出处
期刊:Tumor Biology
[SAGE]
日期:2016-08-03
卷期号:37 (10): 13961-13971
被引量:73
标识
DOI:10.1007/s13277-016-5274-9
摘要
Glioblastomas are deadly neoplasms resistant to current treatment modalities. Fibroblast activation protein (FAP) is a protease which is not expressed in most of the normal adult tissues but is characteristically present in the stroma of extracranial malignancies. FAP is considered a potential therapeutic target and is associated with a worse patient outcome in some cancers. The FAP localization in the glioma microenvironment and its relation to patient survival are unknown. By analyzing 56 gliomas and 15 non-tumorous brain samples, we demonstrate increased FAP expression in a subgroup of high-grade gliomas, in particular on the protein level. FAP expression was most elevated in the mesenchymal subtype of glioblastoma. It was neither associated with glioblastoma patient survival in our patient cohort nor in publicly available datasets. FAP was expressed in both transformed and stromal cells; the latter were frequently localized around dysplastic blood vessels and commonly expressed mesenchymal markers. In a mouse xenotransplantation model, FAP was expressed in glioma cells in a subgroup of tumors that typically did not express the astrocytic marker GFAP. Endogenous FAP was frequently upregulated and part of the FAP+ host cells coexpressed the CXCR4 chemokine receptor. In summary, FAP is expressed by several constituents of the glioblastoma microenvironment, including stromal non-malignant mesenchymal cells recruited to and/or activated in response to glioma growth. The limited expression of FAP in healthy tissues together with its presence in both transformed and stromal cells suggests that FAP may be a candidate target for specific delivery of therapeutic agents in glioblastoma.
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