作者
Eva Nóvoa,Natália da Silva Lima,Maria J. González-Rellan,Maria D.P. Chantada-Vazquez,Joanne Verheij,Amaia Rodrı́guez,Eva M. Esquinas-Román,Marcos F. Fondevila,Mirja Koning,Uxia Fernandez,Alba Cabaleiro,Tamara Parracho,Jose Iglesias-Moure,Samuel Seoane,Begoña Porteiro,Adriana Escudero,Ana Senra,Román Pérez-Fernández,Miguel López,Miguel Fidalgo,Diana Guallar,Maria Luz Martínez‐Chantar,Carlos Diéguez,Ashwin Woodhoo,Vincent Prévot,Markus Schwaninger,Abraham S. Meijnikman,Susana B. Bravo,Gema Frühbeck,Rubén Nogueiras
摘要
Mitochondrial antiviral signaling protein (MAVS) is a critical regulator that activates the host's innate immunity against RNA viruses, and its signaling pathway has been linked to the secretion of proinflammatory cytokines. However, the actions of MAVS on inflammatory pathways during the development of metabolic dysfunction-associated steatotic liver disease (MASLD) have been little studied.