Single cell RNA sequencing (scRNA-Seq) deciphering pathological alterations in streptozotocin-induced diabetic retinas

病态的 生物 细胞 细胞生物学 RNA序列 链脲佐菌素 糖尿病 核糖核酸 生物信息学 计算生物学 医学 病理 遗传学 内分泌学 基因 基因表达 转录组
作者
Licheng Sun,Ruonan Wang,Guangyi Hu,Huazhen Liu,Kangjia Lv,Yi Duan,Ning Shen,Jiali Wu,Jing Hu,Yujuan Liu,Qihuang Jin,Fang Zhang,Xun Xu
出处
期刊:Experimental Eye Research [Elsevier BV]
卷期号:210: 108718-108718 被引量:44
标识
DOI:10.1016/j.exer.2021.108718
摘要

Diabetic retinopathy (DR) is an irreversible and progressive diabetic complication leading to visual impairment, even blindness. Due to the delicate and complicated structure of the retina, the pathology of DR has not been completely elucidated yet. We constructed a transcriptome atlas of >14,000 single cells from healthy and streptozotocin (STZ)-induced diabetic murine retinas to decipher pathological alterations of DR. We found four stress-inducible genes Cirbp, Rmb3, Mt1 and Mt2 commonly induced in most types of retinal cells. Bipolar cells were little affected on both number and gene expression. Diabetes increased expression of inflammatory factor genes in retinal microglia, and stimulated expression of immediate early genes (IEGs) in retinal astrocytes. A large number of genes were deregulated in diabetic vascular endothelial cells (ECs), and the differentially expressed genes were paired to the pathways functioning in metabolism, shear stress and vascular permeability. These pathways were mapped by more deregulated genes in a subpopulation of ECs specifically presented in diabetic retinas (diabetic retinal ECs, DRECs). Moreover, several inflammation pathways were activated in DRECs, and the most significant one is the IL-17 signaling pathway. According to the EC markers, DRECs were mainly capillary ECs, confirmed by immunofluorescent staining of S100a9, a target gene of the IL-17 signaling pathway. This study deciphered pathological alterations of DR, and provided clues for potential targets for DR therapy.
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