糖异生
糖原分解
糖原
脂肪组织
变构调节
脂解
酮发生
化学
效应器
内科学
内分泌学
生物化学
生物
酶
新陈代谢
医学
酮体
作者
Mulchand S. Patel,Robert A. Harris
出处
期刊:Elsevier eBooks
[Elsevier]
日期:2022-07-22
卷期号:: 353-365
标识
DOI:10.1016/b978-0-12-821618-7.00147-4
摘要
Storage of fuels in the fed state followed by their use in the fasted state requires metabolic flexibility. In the fed state, increased substrate supply and insulin promote the synthesis and storage of glycogen in the liver and fat in the adipose tissue. This is achieved by allosteric effectors, covalent modifications of enzymes, and gene transcription that favor glycogen and fat synthesis. In the fasted state, increased levels of glucagon promote lipolysis in the adipose tissue and glycogenolysis, gluconeogenesis, and ketogenesis in the liver. This is achieved by opposite changes in allosteric effectors, covalent modifications of enzymes, and gene transcription.
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