巨噬细胞极化
血管生成
伤口愈合
免疫系统
炎症
癌症研究
PI3K/AKT/mTOR通路
细胞生物学
微泡
M2巨噬细胞
化学
巨噬细胞
免疫学
医学
信号转导
生物
体外
生物化学
小RNA
基因
作者
Xiaoqi Jiang,Junping Ma,Kaikai Xue,Jing Wang,Yu Zhang,Guojian Zhang,Kangyan Wang,Zhe Yao,Qing Hu,Cai Lin,Bo Lei,Cong Mao
出处
期刊:ACS Nano
[American Chemical Society]
日期:2024-01-25
卷期号:18 (5): 4269-4286
被引量:5
标识
DOI:10.1021/acsnano.3c09721
摘要
The repair of diabetic wounds remains challenging, primarily due to the high-glucose-derived immune inhibition which often leads to the excessive inflammatory response, impaired angiogenesis, and heightened susceptibility to infection. However, the means to reduce the immunosuppression and regulate the conversion of M2 phenotype macrophages under a high-glucose microenvironment using advanced biomaterials for diabetic wounds are not yet fully understood. Herein, we report two-dimensional carbide (MXene)-M2 macrophage exosome (Exo) nanohybrids (FM-Exo) for promoting diabetic wound repair by overcoming the high-glucose-derived immune inhibition. FM-Exo showed the sustained release of M2 macrophage-derived exosomes (M2-Exo) up to 7 days and exhibited broad-spectrum antibacterial activity. In the high-glucose microenvironment, relative to the single Exo, FM-Exo could significantly induce the optimized M2a/M2c polarization ratio of macrophages by activating the PI3K/Akt signaling pathway, promoting the proliferation, migration of fibroblasts, and angiogenic ability of endothelial cells. In the diabetic full-thickness wound model, FM-Exo effectively regulated the polarization status of macrophages and promoted their transition to the M2 phenotype, thereby inhibiting inflammation, promoting angiogenesis through VEGF secretion, and improving proper collagen deposition. As a result, the healing process was accelerated, leading to a better healing outcome with reduced scarring. Therefore, this study introduced a promising approach to address diabetic wounds by developing bioactive nanomaterials to regulate immune inhibition in a high-glucose environment.
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