Corrigendum to “Lipocalin-2 mediates non-alcoholic steatohepatitis by promoting neutrophil-macrophage crosstalk via the induction of CXCR2”

Lipocalin-2 mediates non-alcoholic steatohepatitis by promoting neutrophil-macrophage crosstalk via the induction of CXCR2Journal of HepatologyVol. 65Issue 5PreviewNon-alcoholic steatohepatitis (NASH) characterized by co-existence of hepatic steatosis, inflammation and varying degrees of fibrosis is the advanced stage of non-alcoholic fatty liver disease (NAFLD), which is closely associated with a cluster of obesity-related cardio-metabolic complications [1]. Enhanced hepatic infiltration of inflammatory cells, such as hepatic macrophages (Kupffer cells), neutrophils (Lymphocyte antigen 6 complex locus G6D [Ly6G+]), monocytes, CD3+ CD56+ natural killer T cells, is one of key histological features of NASH [2,3]. Full-Text PDF An error was introduced in Fig. 6C of the original manuscript. During preparation of Fig. 6C, the representative image of "HFHC + Rm-LCN2-CXCR2 KO" group was inadvertently duplicated in lieu of the representative image for "SC-CXCR2 KO" group. The corrected immunohistochemistry images shown in the updated Fig. 6, are provided below. The authors apologize for any inconvenience caused.