神经保护
氧化应激
机制(生物学)
冲程(发动机)
脂质过氧化
缺血性中风
病理生理学
神经科学
程序性细胞死亡
医学
生物信息学
药理学
生物
缺血
细胞凋亡
内科学
遗传学
机械工程
认识论
工程类
哲学
作者
A. Gowtham,Chandan Chauhan,Vikrant Rahi,Ravinder K. Kaundal
标识
DOI:10.1080/14728222.2024.2446319
摘要
Introduction Ischemic stroke (IS), a major cause of mortality and disability worldwide, remains a significant healthcare challenge due to limited therapeutic options. Ferroptosis, a distinct iron-dependent form of regulated cell death characterized by lipid peroxidation and oxidative stress, has emerged as a crucial mechanism in IS pathophysiology. This review explores the role of ferroptosis in IS and its potential for driving innovative therapeutic strategies.
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